Part 0: A Rational Assessment of the Primary Causes of Perceptual Symptoms
Summary
This article summarizes my current understanding of the induced symptoms involved in human experimentation and Anomalous Health Incidents (AHI), based on the events I have experienced, my past analysis, and relevant reference materials.
In this part, I clarify that the primary cause of the various symptoms I perceive on a daily basis is not pathological, but rather artificially induced by external means.


1. Introduction
Although I am still able to go outside and continue posting on social media, both my mind and body remain in a state of extreme exhaustion.
Under such circumstances, I continue to speak out about the human rights violations I am being subjected to—violations that are beyond what most people can easily imagine.
Since March 5, 2018—when I was subjected to psychological disruption using sound and light at a hotel operated by a subsidiary of the private railway company Sotetsu, and at a nearby Lawson convenience store—I have continuously perceived some form of torture-like or experimental interference every single day.
For details on the suspicious events I have experienced, please refer to my previously published chronological report.
Naturally, I understand that these claims are difficult for a third party to believe.

2. Symptoms and the Autonomic Nervous System
Some of the symptoms I perceive—such as tinnitus and severe fatigue—can appear similar to those seen in autonomic nervous system disorders.
However, in major English-language medical literature, tinnitus is not classified as a representative symptom of autonomic dysfunction.
While correlations between stress and tinnitus are sometimes discussed, a causal relationship has not been established, and tinnitus is primarily regarded as a “sensory and neural system abnormality.”
Therefore, my tinnitus cannot be sufficiently explained solely by autonomic imbalance.
<Added on November 26, 2025>
Coincidentally, an NHK program introduced a simple method for checking autonomic nervous system function.
I tried the method myself and have posted the results as a supplementary article.
In addition, I have already demonstrated that hearing loss—the most common cause of tinnitus—can be ruled out in my case.

3. Symptoms Synchronized With Specific Actions or Situations
I do not deny the possibility that my autonomic nervous system may not be functioning normally.
However, there are clear patterns that cannot be explained by autonomic dysfunction alone.
My tinnitus and other symptoms change synchronously and repeatedly under specific conditions:
Tinnitus suddenly intensifies the moment I lie down in bed
Severe genital pain or anal pressure occurs simultaneously
These symptoms recur each time I lie down for several consecutive days
Conversely, the symptoms almost completely stop when I begin cooking
Marked cognitive disruption occurs particularly when I attempt to deepen my understanding of technical or analytical topics
These patterns do not match typical autonomic nervous system behavior.

4. Conclusion
Of course, because these symptoms are perceived subjectively, complete objectivity is inherently limited.
However, although the symptoms are not constant, the following factors are consistently observed:
Reproducibility and consistency in how the symptoms manifest
Strength and onset shifting in clear synchronization with specific behaviors or contexts
Correlation with international medical literature regarding the classification of tinnitus
Taking these together, it is more reasonable to conclude that the primary cause of the suffering I continue to perceive is not pathological, but rather artificially induced by external means.

5. Position of This Article
This part (Part 0) serves to establish the premise that the symptoms are artificially induced.
In the next parts (Part 1 and onward), I will examine:
“How such artificial induction could be technically possible.”
The article posted on October 16, 2025 (Part 1) will be significantly revised soon.
<Added on November 26, 2025>
Editor’s Note (February 16, 2026)
On November 25, 2025, Part 0-(2) was published as a supplementary article to Part 0.
Following the publication of the revised Part 1, the original version of Part 1 was made private on February 8, 2026.

